Isilonda esiswini nesilonda se-duodenal (i-peptic ulcer)

Isilonda esiswini nesilonda se-duodenal (i-peptic ulcer)

Thei-peptic ulcer, ebizwa nangokuthi i-gastric ulcer uma itholakala esiswini futhi ibizwa isilonda se-duodenal lapho kwakheka in the duodenum (ingxenye yokuqala emathunjini amancane), kukhona ngandlela-thile amanxeba uhlobo lokuguguleka olungena lujule odongeni lomgudu wokugaya ukudla (bheka umdwebo).

Lezi zilonda zivame ukuba buhlungu: zingena ngqo xhumene nei-asidi ekhona emgudwini wokugaya ukudla. Isimo esiqathaniswa nokusebenzisa i-alcohol swab ekuqaleni.

Inkulumo ethi " i-peptic ulcer »Kuhlanganisa, ngenxa yokufana kokubonakala kwazo, isilonda esiswini kanye nesilonda se-duodenal

Kulinganiselwa ukuthi cishe u-10% wabantu bamazwe athuthukile kungenzeka baphathwe yisilonda ngesinye isikhathi. Abadala be 40 nangaphezulu yizona ezithinteka kakhulu. Izilonda ze-Duodenal zivame izikhathi ezingu-10 kunezilonda zesisu.

Izimbangela

La ibhaktheriya Helicobacter pylori (H. Pylori), igciwane elisinda ku-acidity, liyimbangela eyinhloko yezilonda: kucatshangwa ukuthi libangela cishe amaphesenti angu-60 kuya kwangu-80 izilonda zesisu kanye nama-80% kuya kuma-85%. izilonda ze-duodenal. Lawa magciwane ahlasela ungqimba lwamafinyila ngokuvamile avikela isisu namathumbu amancane ekubeni ne-asidi, futhi kukholakala ukuthi aphazamisa le ndlela yokuzivikela kwabanye abantu. Emazweni athuthukile, u-20% wabantu abaneminyaka engu-40 nangaphansi banaleli gciwane emgudwini wabo wokugaya ukudla. Ingxenye efinyelela ku-50% kulabo abangaphezu kweminyaka engama-60. Cishe amaphesenti angama-20 abathwali bamagciwane azoba nesilonda ngesikhathi sokuphila kwabo.

Ukuthathaanti-inflammatory Izidakamizwa ezingezona steroidal noma ama-NSAID (ngokwesibonelo, i-Aspirin, i-Advil® ne-Motrin®), iyimbangela yesibili evamile yesilonda emgudwini wokugaya ukudla. Inhlanganisela yokutheleleka ngebhaktheriya H. Pylori futhi ukuthatha izidakamizwa eqeda ukuvuvukala synergistically kwandisa ingozi izilonda. Ingozi ibe nkulu ngokuphindwe ka-60.

Nazi ezinye izimbangela:

  • A ukukhiqizwa kwe-asidi ngokweqile ngesisu (i-gastric hyperacidity), okubangelwa ukubhema, ukuphuza ngokweqile, ukucindezeleka okukhulu, ufuzo, njll. Nokho, lezi kungaba yizici ezibhebhethekisayo kunokuba kube izimbangela zangempela zezilonda.
  • A ukusha okukhulu, ukulimala okubalulekile noma ngisho nokucindezeleka ngokomzimba okuhambisana nakho ukugula okungathi sína. Lokhu kudala “izilonda zokucindezeleka”, ezivame ukuba maningi futhi zivame ukutholakala esiswini, ngezinye izikhathi ekuqaleni kwamathumbu amancane (ku-proximal duodenum).
  • Kuyaqabukela, isilonda esiswini siphenduke umdlavuza wesisu onezilonda.

Ama-Acid nama-antacids emgudwini wokugaya ukudla

Odongeni lweisisu, izindlala zikhiqiza amajusi esisu anomthelela ukugaya :

  • we ama-enzyme wokugaya ukudla, njenge- i-pepsine, ehlukanisa amaprotheni abe ama-molecule amancane, ama-peptide;
  • ye 'i-hydrochloric acid (HCL), i-asidi eqinile evumela ama-enzyme okugaya ukuba asebenze futhi abhubhise amagciwane amaningi (ama-parasite, amagciwane, amabhaktheriya, isikhunta) abengangena esiswini.

Okuphakathi kwesisu kusekhona i-asidi. I-pH yayo ihluka ukusuka ku-1,5 kuye ku-5, kuye ngokudla okumunyisiwe futhi kuya ngomuntu ngamunye.

Ezinye izindlala zikhipha amafinyila ahloselwe wona ukuvikela izindonga zangaphakathi zesisu:

  • ce isikhwehlela ivimbela ama-enzyme okugaya ukudla kanye ne-hydrochloric acid ekubhubhiseni ulwelwesi lwesisu ngokwenza ifilimu evikelayo.

Udonga lweamathumbu amancane iphinde ihlanganiswe ne-a ungqimba lwamafinyila okuyivikela ku-acidity ye-chyme, igama elinikezwa "iphalishi lokudla" elivela esiswini.

Evolution

Ngokuvamile isilonda ibonakala kancane kancane emavikini ambalwa. Ingase futhi ibonakale ngokushesha, ngemva kwezinsuku ezimbalwa zokuthatha izidakamizwa ezingezona ukuvuvukala, isibonelo, kodwa lesi simo asivamile kakhulu.

Izinga le- ukuphulukiswa okuzenzakalelayo kungaba cishe ku-40% (esikhathini esiyinyanga engu-1), ikakhulukazi uma isilonda sibangelwa ukuphuza ama-NSAID futhi ayayekwa. Ukuphulukisa okuzenzakalelayo, ngaphandle kokuphinda, kuyivelakancane nokho. Ababhemayo banamathuba amaningi okuthi baphinde baqale kabusha kunabangabhemi.

Uma isilonda singelashwa noma imbangela ingalungiswa, maningi amathuba okuthi izilonda zizophinde zivele ungakapheli unyaka. Kodwa ngisho nokwelashwa okuhle, kukhona ukuphindaphinda ku-20-30% yamacala.

Izinkinga ezingenzeka

Izinkinga azivamile uma kuqhathaniswa. THE'isilonda kungadala i- ukubola kwesisu : igazi libe seligeleza ngaphakathi komgudu wokugaya ukudla. Ukopha ngezinye izikhathi kuba kukhulu, nokuhlanza igazi elibomvu noma elifana nelikabhontshisi wekhofi, elinegazi esitulweni okungenzeka libe bomvu noma libe mnyama. Ukopha nakho kungase kuthule futhi kuhambe kancane. Ungase uqaphele noma ungaqapheli ukuthi indle iba mnyama. Ngempela, ngaphansi kwethonya lamajusi okugaya, igazi liphenduka libe mnyama. Ukopha kungabangela i-anemia ngokuhamba kwesikhathi uma ingabonwa. Uphawu lokuqala lwesilonda kungase kube ukopha, ngaphandle kobuhlungu ngaphambili, ikakhulukazi kubantu asebekhulile. Kufanele uthintane nodokotela ngaphandle kokulibala.

Enye inkinga, engavamile kakhulu kunokopha, i i-perforation udonga oluphelele lomgudu wokugaya ukudla. Lesi simo sibangela ubuhlungu besisu obunobudlova, obuphuthuma ngokushesha ku-peritonitis. Lesi yisimo esiphuthumayo sezokwelapha nesokuhlinzwa.

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